Home Compounds Immune & Anti-Aging LL-37
Immune & Anti-Aging Research Moderate Evidence

LL-37

Only cathelicidin antimicrobial peptide in humans. Studied for broad-spectrum antimicrobial, immunomodulatory, wound healing, and potential anticancer properties.

antimicrobialcathelicidinimmunewound healingantibacterialanticancer
Half-life
Very short; rapidly degraded by proteases
SKUs
1
Evidence
Moderate Evidence

LL-37 is a human cathelicidin antimicrobial peptide — the only cathelicidin made by the human body. It is produced by immune cells and epithelial cells as part of the innate immune response. It has both direct antimicrobial activity and immune-modulating properties. Research has studied it in infection, wound healing, inflammatory conditions, and cancer biology.

Antimicrobial Research
LL-37 was originally identified for its ability to kill bacteria, fungi, viruses, and parasites by disrupting their cell membranes. Research has characterized its activity against drug-resistant bacteria, which has generated interest in it as a potential template for new antimicrobial compounds.
Wound Healing Research
LL-37 is produced at wound sites and plays a role in the healing response. Research has examined how LL-37 promotes cell migration, angiogenesis, and re-epithelialization at wounds. Some clinical interest has developed in topical LL-37 formulations for chronic wound management.
Immune Modulation Research
Beyond direct antimicrobial activity, LL-37 modulates the immune response in complex ways. It can be pro-inflammatory in some contexts (recruiting immune cells) and anti-inflammatory in others (modulating cytokine production). This dual role has implications for its research in inflammatory diseases.
Cancer Research
Research has examined LL-37's roles in cancer — sometimes appearing to inhibit tumor growth through immune stimulation, other times appearing to promote tumor progression in certain cancer types. Its biology in cancer contexts is complex and not yet fully resolved.
  • Broad antimicrobial activity against bacteria, fungi, and some viruses in laboratory studies.
  • Promotes wound healing through multiple mechanisms including angiogenesis and cell migration.
  • Complex immune-modulating effects that are context-dependent.
  • Mixed findings in cancer research — inhibitory in some contexts, potentially promotional in others.
  • Limited formal human clinical trial data for therapeutic applications.

LL-37 has a complex biology that makes it difficult to use as a straightforward therapeutic agent. Its immune effects are bidirectional depending on context. The cancer biology findings are concerning enough that unsupervised systemic use raises questions. Human therapeutic trials are limited. It is not FDA-approved for any application.

LL-37 is produced by your skin, lungs, and immune cells as a first responder against infections. It works by inserting itself into the membranes of bacteria and disrupting them — essentially punching holes in the invader's outer layer. It also sends signals to immune cells, telling them to come to the site of infection or injury. At wound sites, these signals also kick off healing processes by telling cells to grow and new blood vessels to form. LL-37's activity is not as simple as just killing pathogens — it is more like a multipurpose molecular alarm system that can turn up or dial down different aspects of the immune response depending on what is happening around it. This complexity makes it both interesting for research and challenging to develop as a drug.

LL-37 has not been evaluated in large human clinical trials. The cancer research findings — suggesting it may promote certain cancer types in some contexts — are a meaningful theoretical concern for systemic use. At inflammatory sites it can increase pro-inflammatory signaling. It is not FDA-approved for any use. The complexity of its biology makes its safety profile in diverse populations difficult to predict without more clinical research.

Moderate Evidence

This compound has been studied in Phase 1 or Phase 2 human trials. Evidence is encouraging but more large-scale trials are needed.

Published Research Ranges
Primarily topical research; systemic studies limited
Research Context Only: These are ranges reported in published scientific studies for educational reference. They are not dosing recommendations. This is not medical advice. Always consult a qualified healthcare professional.

Sources listed here are from the platform research library. All links open the original publication. No citations are generated by AI.

The Cathelicidin Antimicrobial Peptide LL-37 Modulates Epithelial and Leukocyte Functions
Nature Reviews Immunology • 2006  • DOI: 10.1038/nri1894
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LL-37 and Its Role in Wound Healing and Skin Immunity
Journal of Investigative Dermatology • 2009  • DOI: 10.1038/jid.2008.448
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Human Cathelicidin LL-37 Is a Multifunctional Peptide with Antimicrobial and Immunomodulatory Activities
Cellular Microbiology • 2004  • DOI: 10.1111/j.1462-5822.2004.00408.x
View Source
LL-37 Improves Chronic Wound Healing: Clinical Study
PLOS ONE • 2012  • DOI: 10.1371/journal.pone.0052555
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The Amyloid-β Pathway in Alzheimer's Disease
 • 2021  • DOI: 10.1038/s41380-021-01249-0
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Long non-coding RNAs: definitions, functions, challenges and recommendations
 • 2023  • DOI: 10.1038/s41580-022-00566-8
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Chronic wounds
 • 2022  • DOI: 10.1038/s41572-022-00377-3
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Neutrophils in chronic inflammatory diseases
 • 2022  • DOI: 10.1038/s41423-021-00832-3
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Antimicrobial peptides: mechanism of action, activity and clinical potential
 • 2021  • DOI: 10.1186/s40779-021-00343-2
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Diabetic Wound-Healing Science
 • 2021  • DOI: 10.3390/medicina57101072
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Interaction between microbiota and immunity in health and disease
 • 2020  • DOI: 10.1038/s41422-020-0332-7
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Antimicrobial Peptides: Classification, Design, Application and Research Progress in Multiple Fields
 • 2020  • DOI: 10.3389/fmicb.2020.582779
View Source
Staphylococcal Biofilm Development: Structure, Regulation, and Treatment Strategies
 • 2020  • DOI: 10.1128/mmbr.00026-19
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Coronavirus infections and immune responses
 • 2020  • DOI: 10.1002/jmv.25685
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Evidence that Vitamin D Supplementation Could Reduce Risk of Influenza and COVID-19 Infections and Deaths
 • 2020  • DOI: 10.3390/nu12040988
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Emerging Strategies to Combat ESKAPE Pathogens in the Era of Antimicrobial Resistance: A Review
 • 2019  • DOI: 10.3389/fmicb.2019.00539
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The antimicrobial peptides and their potential clinical applications
 • 2019
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Skeletal and Extraskeletal Actions of Vitamin D: Current Evidence and Outstanding Questions
 • 2019  • DOI: 10.1210/er.2018-00126
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What is the Healthy Gut Microbiota Composition? A Changing Ecosystem across Age, Environment, Diet, and Diseases
 • 2019  • DOI: 10.3390/microorganisms7010014
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Tumor Microenvironment
 • 2019  • DOI: 10.3390/medicina56010015
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Strategies for combating bacterial biofilms: A focus on anti-biofilm agents and their mechanisms of action
 • 2018  • DOI: 10.1080/21505594.2017.1313372
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Skin microbiota-host interactions
 • 2018  • DOI: 10.1038/nature25177
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Neutrophils in tissue injury and repair
 • 2018  • DOI: 10.1007/s00441-017-2785-7
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Targeting microbial biofilms: current and prospective therapeutic strategies
 • 2017  • DOI: 10.1038/nrmicro.2017.99
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Polymyxins: Antibacterial Activity, Susceptibility Testing, and Resistance Mechanisms Encoded by Plasmids or Chromosomes
 • 2017  • DOI: 10.1128/cmr.00064-16
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Antioxidant and anti-inflammatory effects of zinc. Zinc-dependent NF-κB signaling
 • 2017  • DOI: 10.1007/s10787-017-0309-4
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Antimicrobial Peptides: Mechanisms of Action and Resistance
 • 2017  • DOI: 10.1177/0022034516679973
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Tumour-associated macrophages as treatment targets in oncology
 • 2017  • DOI: 10.1038/nrclinonc.2016.217
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Microglial M1/M2 polarization and metabolic states
 • 2016  • DOI: 10.1111/bph.13139
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Antimicrobial Peptides: An Emerging Category of Therapeutic Agents
 • 2016  • DOI: 10.3389/fcimb.2016.00194
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Cancer chemotherapy and beyond: Current status, drug candidates, associated risks and progress in targeted therapeutics
 • 2023  • DOI: 10.1016/j.gendis.2022.02.007
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Signaling pathways and targeted therapies for psoriasis
 • 2023  • DOI: 10.1038/s41392-023-01655-6
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Immunomodulatory actions of vitamin D in various immune-related disorders: a comprehensive review
 • 2023  • DOI: 10.3389/fimmu.2023.950465
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Methicillin Resistant Staphylococcus aureus: Molecular Mechanisms Underlying Drug Resistance Development and Novel Strategies to Combat
 • 2023  • DOI: 10.2147/idr.s428103
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Engineering mesoporous silica nanoparticles for drug delivery: where are we after two decades?
 • 2022  • DOI: 10.1039/d1cs00659b
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Vitamin D and Cancer: An Historical Overview of the Epidemiology and Mechanisms
 • 2022  • DOI: 10.3390/nu14071448
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Stem cell-based therapy for human diseases
 • 2022  • DOI: 10.1038/s41392-022-01134-4
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Impact of gut microbiome on skin health: gut-skin axis observed through the lenses of therapeutics and skin diseases
 • 2022  • DOI: 10.1080/19490976.2022.2096995
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Neutrophils in cancer carcinogenesis and metastasis
 • 2021  • DOI: 10.1186/s13045-021-01187-y
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Toll-like receptor 3 (TLR3) regulation mechanisms and roles in antiviral innate immune responses
 • 2021  • DOI: 10.1631/jzus.b2000808
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Membrane-disruptive peptides/peptidomimetics-based therapeutics: Promising systems to combat bacteria and cancer in the drug-resistant era
 • 2021  • DOI: 10.1016/j.apsb.2021.07.014
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Exosomes: Potential Disease Biomarkers and New Therapeutic Targets
 • 2021  • DOI: 10.3390/biomedicines9081061
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Inflammatory Bowel Disease-Associated Colorectal Cancer: Translational Risks from Mechanisms to Medicines
 • 2021  • DOI: 10.1093/ecco-jcc/jjab102
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The Influence of Nutritional Factors on Immunological Outcomes
 • 2021  • DOI: 10.3389/fimmu.2021.665968
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A comprehensive review of the functions of YB-1 in cancer stemness, metastasis and drug resistance
 • 2021  • DOI: 10.1016/j.cellsig.2021.110073
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Rational Design of Immunomodulatory Hydrogels for Chronic Wound Healing
 • 2021  • DOI: 10.1002/adma.202100176
View Source
Antimicrobial Peptides: A New Hope in Biomedical and Pharmaceutical Fields
 • 2021  • DOI: 10.3389/fcimb.2021.668632
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Therapeutic host-directed strategies to improve outcome in tuberculosis
 • 2020  • DOI: 10.1038/s41385-019-0226-5
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Antimicrobial peptides - Advances in development of therapeutic applications
 • 2020  • DOI: 10.1016/j.lfs.2020.118407
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Targeting Biofilms Therapy: Current Research Strategies and Development Hurdles
 • 2020  • DOI: 10.3390/microorganisms8081222
View Source
Significance of LL-37 on Immunomodulation and Disease Outcome
 • 2020  • DOI: 10.1155/2020/8349712
View Source
The Immune Functions of Keratinocytes in Skin Wound Healing
 • 2020  • DOI: 10.3390/ijms21228790
View Source
T cell pathology in skin inflammation
 • 2019  • DOI: 10.1007/s00281-019-00742-7
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The Dynamics of the Skin's Immune System
 • 2019  • DOI: 10.3390/ijms20081811
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Pathophysiology of atopic dermatitis: Clinical implications
 • 2019  • DOI: 10.2500/aap.2019.40.4202
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<i>Staphylococcus aureus</i> Secreted Toxins and Extracellular Enzymes
 • 2019  • DOI: 10.1128/microbiolspec.gpp3-0039-2018
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More friend than foe: the emerging role of neutrophils in tissue repair
 • 2019  • DOI: 10.1172/jci124616
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Membrane Active Peptides and Their Biophysical Characterization
 • 2018  • DOI: 10.3390/biom8030077
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Mechanisms and Targeted Therapies for Pseudomonas aeruginosa Lung Infection
 • 2018  • DOI: 10.1164/rccm.201705-1043so
View Source
Plasma Gelsolin: Indicator of Inflammation and Its Potential as a Diagnostic Tool and Therapeutic Target
 • 2018  • DOI: 10.3390/ijms19092516
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60 sources · Platform research library · Not generated by AI

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Research Education Only: This profile is for educational purposes only. All information is sourced from published scientific literature. This is not medical advice. Not for human consumption. Consult qualified medical professionals for any health decisions.