Home Compounds GLP-1 & Metabolic Retatrutide
GLP-1 & Metabolic Research High Evidence

Retatrutide

Triple agonist (GIP/GLP-1/Glucagon) under phase 2/3 investigation. Early trials show remarkable metabolic and weight-reduction signals.

triple agonistglp-1gipglucagonmetabolicobesity
Half-life
6 days
SKUs
8
Evidence
High Evidence

Retatrutide is a once-weekly injectable peptide that activates three gut and metabolic hormone receptors simultaneously — GLP-1, GIP, and glucagon. No compound in the GLP-1 class had targeted all three at once in human trials before it. Developed by Eli Lilly, it showed the highest weight reduction percentages recorded in a peptide-based clinical trial when its phase 2 data were published in 2023, generating significant research and medical interest.

Weight Management Research
The phase 2 JAMA trial published in 2023 tested retatrutide in adults with obesity. The highest dose group (12mg weekly) showed a mean weight reduction of 24.2% at 48 weeks — the highest recorded in a peptide-based obesity trial at that time. The trial was randomized and placebo-controlled.
Type 2 Diabetes
Phase 2 trials also examined retatrutide in type 2 diabetes patients, showing significant HbA1c reductions consistent with GLP-1 receptor activation alongside the weight effects.
NASH and Liver Research
The liver stores excess fat when the body's metabolic signals are out of balance, and glucagon is one of the hormones involved in telling the liver to burn that fat. Retatrutide's glucagon component may make it useful for non-alcoholic fatty liver disease — a condition where excess fat builds up in liver tissue and can lead to inflammation over time. Research examining retatrutide specifically for this indication is underway.
Cardiovascular Research
Phase 3 trials are now underway including cardiovascular outcomes research, following the established pattern of GLP-1 class development where metabolic benefits are followed by cardiovascular trials.
  • Phase 2 trial: 24.2% mean weight reduction at 48 weeks at the highest dose — highest in a peptide obesity trial at time of publication.
  • Significant HbA1c reductions in type 2 diabetes patients.
  • Dose-dependent effects across GLP-1, GIP, and glucagon pathways.
  • Phase 3 trials underway.
  • Not yet FDA-approved.

Retatrutide has not completed phase 3 trials. Phase 2 data, while striking, do not constitute approval evidence. GI side effects were common — nausea, vomiting, diarrhea — consistent with the GLP-1 class and amplified by the additional glucagon component. The glucagon component also produces a modest increase in resting heart rate — a known effect of glucagon receptor activation that was monitored in the trials and is factored into ongoing phase 3 safety assessments. Long-term data beyond 48 weeks are not available from published trials.

Retatrutide activates three receptors at once. GLP-1 receptor activation reduces appetite, slows stomach emptying, and improves insulin secretion. GIP receptor activation appears to enhance the effects of GLP-1, reduce fat storage, and improve metabolic flexibility. Glucagon receptor activation increases the rate at which the liver burns fat and raises energy expenditure — glucagon is normally a counter-regulatory hormone that raises blood sugar, but when balanced with the insulin-supporting effects of GLP-1 and GIP, the glucagon component appears to add significant fat-burning activity without causing problematic blood sugar elevations. The combination of all three effects working simultaneously appears to explain the unusually large weight reduction seen in the trial.

Retatrutide is not FDA-approved. In phase 2 trials the GI side effect profile was consistent with GLP-1 class compounds, with significant nausea during uptitration. Heart rate increases consistent with glucagon receptor activation were observed and require monitoring. Phase 3 long-term safety data are still being collected.

High Evidence

This compound has been studied in multiple large randomized controlled trials with human subjects. The evidence base is strong and consistent across independent research groups.

Published Research Ranges
1–12mg weekly in Phase 2 trials
Research Context Only: These are ranges reported in published scientific studies for educational reference. They are not dosing recommendations. This is not medical advice. Always consult a qualified healthcare professional.

Sources listed here are from the platform research library. All links open the original publication. No citations are generated by AI.

Retatrutide, a GIP, GLP-1 and Glucagon Receptor Agonist, for People with Type 2 Diabetes
The Lancet • 2023  • DOI: 10.1016/S0140-6736(23)01053-X
View Source
Retatrutide Phase 2 Obesity Trial
New England Journal of Medicine • 2023  • DOI: 10.1056/NEJMoa2301972
View Source
Triple hormone receptor agonist retatrutide for metabolic dysfunction-associated steatotic liver disease: a randomized phase 2a trial
 • 2024  • DOI: 10.1038/s41591-024-03018-2
View Source
Efficacy and Safety of Mazdutide in Chinese Patients With Type 2 Diabetes: A Randomized, Double-Blind, Placebo-Controlled Phase 2 Trial
 • 2024  • DOI: 10.2337/dc23-1287
View Source
Triple-Hormone-Receptor Agonist Retatrutide for Obesity - A Phase 2 Trial
 • 2023  • DOI: 10.1056/nejmoa2301972
View Source
Retatrutide, a GIP, GLP-1 and glucagon receptor agonist, for people with type 2 diabetes: a randomised, double-blind, placebo and active-controlled, parallel-group, phase 2 trial conducted in the USA
 • 2023  • DOI: 10.1016/s0140-6736(23)01053-x
View Source
Results from three phase 1 trials of NNC9204-1177, a glucagon/GLP-1 receptor co-agonist: Effects on weight loss and safety in adults with overweight or obesity
 • 2023  • DOI: 10.1016/j.molmet.2023.101801
View Source
LY3437943, a novel triple GIP, GLP-1, and glucagon receptor agonist in people with type 2 diabetes: a phase 1b, multicentre, double-blind, placebo-controlled, randomised, multiple-ascending dose trial
 • 2022  • DOI: 10.1016/s0140-6736(22)02033-5
View Source
Efficacy and Safety of Glucagon-Like Peptide-1 Receptor Agonists for Weight Loss Among Adults Without Diabetes : A Systematic Review of Randomized Controlled Trials
 • 2025  • DOI: 10.7326/annals-24-01590
View Source
Emerging pharmacotherapies for obesity: A systematic review
 • 2025  • DOI: 10.1124/pharmrev.123.001045
View Source
Seven glucagon-like peptide-1 receptor agonists and polyagonists for weight loss in patients with obesity or overweight: an updated systematic review and network meta-analysis of randomized controlled trials
 • 2024  • DOI: 10.1016/j.metabol.2024.156038
View Source
Efficacy and safety of once-weekly tirzepatide for weight management compared to placebo: An updated systematic review and meta-analysis including the latest SURMOUNT-2 trial
 • 2024  • DOI: 10.1007/s12020-024-03896-z
View Source
Comparison of the efficacy and safety of 10 glucagon-like peptide-1 receptor agonists as add-on to metformin in patients with type 2 diabetes: a systematic review
 • 2023  • DOI: 10.3389/fendo.2023.1244432
View Source
A Critical View over the Newest Antidiabetic Molecules in Light of Efficacy-A Systematic Review and Meta-Analysis
 • 2023  • DOI: 10.3390/ijms24119760
View Source
What is the pipeline for future medications for obesity?
 • 2025  • DOI: 10.1038/s41366-024-01473-y
View Source
Advance in peptide-based drug development: delivery platforms, therapeutics and vaccines
 • 2025  • DOI: 10.1038/s41392-024-02107-5
View Source
The Lancet Commission on rethinking coronary artery disease: moving from ischaemia to atheroma
 • 2025  • DOI: 10.1016/s0140-6736(25)00055-8
View Source
Evolving Global Etiology of Hepatocellular Carcinoma (HCC): Insights and Trends for 2024
 • 2025  • DOI: 10.1016/j.jceh.2024.102406
View Source
An overview of obesity-related complications: The epidemiological evidence linking body weight and other markers of obesity to adverse health outcomes
 • 2025  • DOI: 10.1111/dom.16263
View Source
Metabolic dysfunction-associated steatotic liver disease and the heart
 • 2025  • DOI: 10.1097/hep.0000000000000735
View Source
Glucose-dependent insulinotropic polypeptide (GIP)
 • 2025  • DOI: 10.1016/j.molmet.2025.102118
View Source
Type 2 diabetes mellitus - conventional therapies and future perspectives in innovative treatment
 • 2025  • DOI: 10.1016/j.bbrep.2025.102037
View Source
Obesity-Related Chronic Kidney Disease: From Diagnosis to Treatment
 • 2025  • DOI: 10.3390/diagnostics15020169
View Source
Glucagon-like peptide-1 receptor: mechanisms and advances in therapy
 • 2024  • DOI: 10.1038/s41392-024-01931-z
View Source
Efficacy and Safety of GLP-1 Medicines for Type 2 Diabetes and Obesity
 • 2024  • DOI: 10.2337/dci24-0003
View Source
Diabetes mellitus-Progress and opportunities in the evolving epidemic
 • 2024  • DOI: 10.1016/j.cell.2024.06.029
View Source
GLP-1 single, dual, and triple receptor agonists for treating type 2 diabetes and obesity: a narrative review
 • 2024  • DOI: 10.1016/j.eclinm.2024.102782
View Source
The Pharmaceutical Industry in 2023: An Analysis of FDA Drug Approvals from the Perspective of Molecules
 • 2024  • DOI: 10.3390/molecules29030585
View Source
Incretin-Based Weight Loss Pharmacotherapy: Can Resistance Exercise Optimize Changes in Body Composition?
 • 2024  • DOI: 10.2337/dci23-0100
View Source
Activation of human brown adipose tissue by a β3-adrenergic receptor agonist
 • 2015  • DOI: 10.1016/j.cmet.2014.12.009
View Source
Glucagon-Like Peptide-1 Receptor Agonists Improve MASH and Liver Fibrosis: A Meta-Analysis of Randomised Controlled Trials
 • 2025  • DOI: 10.1111/liv.70256
View Source
Effect of a Low-Calorie Dietary Intervention on Liver Health and Body Weight in Adults with Metabolic-Dysfunction Associated Steatotic Liver Disease (MASLD) and Overweight/Obesity: A Systematic Review and Meta-Analysis
 • 2024  • DOI: 10.3390/nu16071030
View Source
Obesity: pathophysiology and therapeutic interventions
 • 2025  • DOI: 10.1186/s43556-025-00264-9
View Source
Resmetirom: The First Disease-Specific Treatment for MASH
 • 2025  • DOI: 10.1155/ije/6430023
View Source
Type 2 Diabetes and the Multifaceted Gut-X Axes
 • 2025  • DOI: 10.3390/nu17162708
View Source
Metabolic Syndrome and Liver Disease: Re-Appraisal of Screening, Diagnosis, and Treatment Through the Paradigm Shift from NAFLD to MASLD
 • 2025  • DOI: 10.3390/jcm14082750
View Source
Advancing Cardiovascular, Kidney, and Metabolic Medicine: A Narrative Review of Insights and Innovations for the Future
 • 2025  • DOI: 10.1007/s13300-025-01738-3
View Source
Therapeutic Targets and Approaches to Manage Inflammation of NAFLD
 • 2025  • DOI: 10.3390/biomedicines13020393
View Source
Drug Pipeline for MASLD: What Can Be Learned from the Successful Story of Resmetirom
 • 2025  • DOI: 10.3390/cimb47030154
View Source
New advances in novel pharmacotherapeutic candidates for the treatment of metabolic dysfunction-associated steatohepatitis (MASH) between 2022 and 2024
 • 2025  • DOI: 10.1038/s41401-024-01466-7
View Source
SGLT2 Inhibitors and GLP-1 Receptor Agonists in Diabetic Kidney Disease: Evolving Evidence and Clinical Application
 • 2025  • DOI: 10.4093/dmj.2025.0220
View Source
Cardiovascular-Kidney-Metabolic Syndrome: A New Paradigm in Clinical Medicine or Going Back to Basics?
 • 2025  • DOI: 10.3390/jcm14082833
View Source
Decoding the Liver-Heart Axis in Cardiometabolic Diseases
 • 2025  • DOI: 10.1161/circresaha.125.325492
View Source
Advancements in pharmacological treatment of NAFLD/MASLD: a focus on metabolic and liver-targeted interventions
 • 2024  • DOI: 10.1093/gastro/goae029
View Source
Pipeline of New Drug Treatment for Non-alcoholic Fatty Liver Disease/Metabolic Dysfunction-associated Steatotic Liver Disease
 • 2024  • DOI: 10.14218/jcth.2024.00123
View Source
The Road towards Triple Agonists: Glucagon-Like Peptide 1, Glucose-Dependent Insulinotropic Polypeptide and Glucagon Receptor - An Update
 • 2024  • DOI: 10.3803/enm.2024.1942
View Source
From liver to hormones: The endocrine consequences of cirrhosis
 • 2024  • DOI: 10.3748/wjg.v30.i9.1073
View Source
Oligonucleotide therapies for nonalcoholic steatohepatitis
 • 2024  • DOI: 10.1016/j.omtn.2024.102184
View Source
Advances in management of metabolic dysfunction-associated steatotic liver disease: from mechanisms to therapeutics
 • 2024  • DOI: 10.1186/s12944-024-02092-2
View Source
Metabolic disease and the liver: A review
 • 2024  • DOI: 10.4254/wjh.v16.i1.33
View Source
Glucagon-like Peptide 1, Glucose-Dependent Insulinotropic Polypeptide, and Glucagon Receptor Agonists in Metabolic Dysfunction-Associated Steatotic Liver Disease: Novel Medication in New Liver Disease Nomenclature
 • 2024  • DOI: 10.3390/ijms25073832
View Source
Drug treatment for metabolic dysfunction-associated steatotic liver disease: Progress and direction
 • 2024  • DOI: 10.1097/cm9.0000000000003355
View Source
Glucagon-Like Peptide-1 Based Therapies: A New Horizon in Obesity Management
 • 2024  • DOI: 10.3803/enm.2024.1940
View Source
Adipocentric origin of the common cardiometabolic complications of obesity in the young up to the very old: pathophysiology and new therapeutic opportunities
 • 2024  • DOI: 10.3389/fmed.2024.1365183
View Source
New Molecules in Type 2 Diabetes: Advancements, Challenges and Future Directions
 • 2024  • DOI: 10.3390/ijms25116218
View Source
Lipid metabolism in MASLD and MASH: From mechanism to the clinic
 • 2024  • DOI: 10.1016/j.jhepr.2024.101185
View Source
Obesity: the perfect storm for heart failure
 • 2024  • DOI: 10.1002/ehf2.14641
View Source
Update on Obesity and Cardiovascular Risk: From Pathophysiology to Clinical Management
 • 2024  • DOI: 10.3390/nu16162781
View Source
GLP-1-based therapies for the treatment of resistant hypertension in individuals with overweight or obesity: a review
 • 2024  • DOI: 10.1016/j.eclinm.2024.102789
View Source

59 sources · Platform research library · Not generated by AI

RT5RT10RT15RT20RT30RT40RT50RT60

Combination use is not endorsed - provided for research context only.

Want More Detailed Research?

Ask the AI anything about Retatrutide - mechanisms, trial summaries, pharmacokinetics, and comparisons.

Ask AI About Retatrutide
Research Education Only: This profile is for educational purposes only. All information is sourced from published scientific literature. This is not medical advice. Not for human consumption. Consult qualified medical professionals for any health decisions.